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American Journal of Clinical Nutrition, Vol 26, 352-361, Copyright © 1973 by The American Society for Clinical Nutrition, Inc.

Calcium metabolism in pancreatic disease

Arthur D'Souza M.B.1 and Martin H. Floch M.S., M.D.1

1 From the Yale Affiliated Gastrointestinal Program; Departments of Medicine, Norwalk Hospital, Norwalk, Connecticut 06850, and Yale University School of Medicine, New Haven, Connecticut

In summary, calcium homeostasis is related to dietary intake, vitamin D, parathormone, thyrocalcitonin, and probably numerous physical intestinal factors and other hormones. It becomes disturbed in pancreatic disease when there is malabsorption of fats and vitamin D. In acute pancreatitis, the serum levels may fall precipitously for as yet unproved reasons, but may be related to development of calcium soaps or glucagon elevations. In chronic pancreatic disease, serum calcium and body stores may fall due to related malabsorption or precipitation of soaps, or both. The correction of hypocalcemia in these states at present is largely concerned with calcium replacement and correction of malabsorption.






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Copyright © 1973 by The American Society for Nutrition