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American Journal of Clinical Nutrition, Vol 27, 743-745, Copyright © 1974 by The American Society for Clinical Nutrition, Inc.

Release by ethanol of vitamins into rat liver perfusates

Michael F. Sorrell M.D.1, Herman Baker Ph.D.1, Anthony J. Barak Ph.D.1, and Oscar Frank Ph.D.1

1 From the Departments of Medicine and Biochemistry, Liver Study Unit, University of Nebraska Medical Center and Veterans Administration Hospital, 4101 Woolworth Street, Omaha, Nebraska 68105, and the Departments of Medicine and Preventive Medicine, College of Medicine and Dentistry of New Jersey-New Jersey Medical School, 88 Ross Street, East Orange, New Jersey 07018

In vitro perfusion of rat liver with ethanol (EtOH) causes a release of all vitamins from the liver stores, except biotin, vitamins A, E, C, and beta-carotene. The concentration of vitamins released from the liver by EtOH in decreasing order was B12 > B6 > pantothenate > thiamin > riboflavin > nicotinate > folate. The B12 concentration in the EtOH perfusate was high when compared with the perfusate without EtOH, implying that B12 can be rapidly released from a liver exposed to EtOH. Results also suggest that when a liver exposed to EtOH releases a vitamin from its stores, it cannot rebind the released vitamin while EtOH is being recirculated.




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H. Baker, C. B. Leevy, B. DeAngelis, O. Frank, and E. R. Baker
Cobalamin (Vitamin B12) and Holotranscobalamin Changes in Plasma and Liver Tissue in Alcoholics with Liver Disease
J. Am. Coll. Nutr., June 1, 1998; 17(3): 235 - 238.
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