AJCN North Carolina Research Campus
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
QUICK SEARCH:   [advanced]


     

This Article
Right arrow Full Text (PDF)
Right arrow Purchase Article
Right arrow View Shopping Cart
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Mezey, E.
Right arrow Articles by Herlong, H. F.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Mezey, E.
Right arrow Articles by Herlong, H. F.
Agricola
Right arrow Articles by Mezey, E.
Right arrow Articles by Herlong, H. F.

American Journal of Clinical Nutrition, Vol 48, 148-151, Copyright © 1988 by The American Society for Clinical Nutrition, Inc

ORIGINAL RESEARCH COMMUNICATIONS

Alcohol and dietary intake in the development of chronic pancreatitis and liver disease in alcoholism

E Mezey, CJ Kolman, AM Diehl, MC Mitchell and HF Herlong
Alcohol Research Center, Johns Hopkins University School of Medicine, Baltimore, MD 21205.

Alcohol and dietary intake were determined in alcoholic patients with chronic pancreatitis and alcoholic liver disease. Patients with chronic pancreatitis, alcoholic hepatitis, and cirrhosis ingested approximately 50% of their calories as alcohol, and all had low mean intakes of protein, carbohydrate, and fat as compared with control subjects. Patients with severe alcoholic hepatitis had the lowest intake of nonalcohol calories and protein. Women with chronic pancreatitis had ingested alcohol for a shorter period of time than men whereas women with alcoholic hepatitis and cirrhosis had ingested less alcohol per kilogram body weight per day as compared with men. This study does not support the hypothesis that consumption of a high-protein and high-fat diet is a factor in the development of chronic pancreatitis in the alcoholic patient. The increased susceptibility of women as compared with men to alcoholic liver disease is established.


This article has been cited by other articles:


Home page
 Alcohol AlcoholHome page
S. Pelletier, E. Vaucher, R. Aider, S. Martin, P. Perney, J. L. Balmes, and B. Nalpas
WINE CONSUMPTION IS NOT ASSOCIATED WITH A DECREASED RISK OF ALCOHOLIC CIRRHOSIS IN HEAVY DRINKERS
Alcohol Alcohol., November 1, 2002; 37(6): 618 - 621.
[Abstract] [Full Text] [PDF]

Home page
 Am. J. Physiol. Gastrointest. Liver Physiol.Home page
A. A. Nanji, K. Jokelainen, M. Fotouhinia, A. Rahemtulla, P. Thomas, G. L. Tipoe, G. L. Su, and A. J. Dannenberg
Increased severity of alcoholic liver injury in female rats: role of oxidative stress, endotoxin, and chemokines
Am J Physiol Gastrointest Liver Physiol, December 1, 2001; 281(6): G1348 - G1356.
[Abstract] [Full Text] [PDF]

Home page
 Toxicol SciHome page
G. M. Pastino and R. B. Conolly
Application of a Physiologically Based Pharmacokinetic Model to Estimate the Bioavailability of Ethanol in Male Rats: Distinction between Gastric and Hepatic Pathways of Metabolic Clearance
Toxicol. Sci., June 1, 2000; 55(2): 256 - 265.
[Abstract] [Full Text] [PDF]

Home page
 Alcohol AlcoholHome page
T. Wetterling, C. Veltrup, M. Driessen, and U. John
DRINKING PATTERN AND ALCOHOL-RELATED MEDICAL DISORDERS
Alcohol Alcohol., May 1, 1999; 34(3): 330 - 336.
[Abstract] [Full Text] [PDF]


HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Copyright © 1988 by The American Society for Nutrition