AJCN North Carolina Research Campus
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
 QUICK SEARCH:   [advanced]


     


This Article
Right arrow Full Text (PDF)
Right arrow Purchase Article
Right arrow View Shopping Cart
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Right arrow Citation Map
Services
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Morales, L. M.
Right arrow Articles by Bonilla, E.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Morales, L. M.
Right arrow Articles by Bonilla, E.
Agricola
Right arrow Articles by Morales, L. M.
Right arrow Articles by Bonilla, E.

American Journal of Clinical Nutrition, Vol 50, 145-150, Copyright © 1989 by The American Society for Clinical Nutrition, Inc


ORIGINAL RESEARCH COMMUNICATIONS

Nutritional evaluation of Huntington disease patients

LM Morales, J Estevez, H Suarez, R Villalobos, L Chacin de Bonilla and E Bonilla
Faculty of Science, Universidad del Zulia, Maracaibo, Venezuela.

A nutritional survey and evaluation was made in Huntington disease patients by the 24-h-recall method. Control subjects and choreic patients consumed a diet that supplied all the essential amino acids. The diet was hypocaloric, rich in animal protein, and low in fat and carbohydrates. The ratio of calcium to phosphorus in the groups studied was less than 1. High vitamin A and low vitamin C and niacin intakes were observed in Huntington disease patients. Only 17% of control subjects showed weight deficiency; 55% of the patients at stages III and IV of the disease were malnourished despite receiving the same food intake as controls. Although iron intake was deficient in all groups studied, it was enough to maintain normal serum levels of this metal. The deficiencies found in some nutrients do not explain the clinical manifestations observed in Huntington disease patients.


This article has been cited by other articles:


Home page
Hum Mol GenetHome page
J. M. Van Raamsdonk, W. T. Gibson, J. Pearson, Z. Murphy, G. Lu, B. R. Leavitt, and M. R. Hayden
Body weight is modulated by levels of full-length Huntingtin
Hum. Mol. Genet., May 1, 2006; 15(9): 1513 - 1523.
[Abstract] [Full Text] [PDF]


Home page
Hum Mol GenetHome page
A. D. Strand, A. K. Aragaki, D. Shaw, T. Bird, J. Holton, C. Turner, S. J. Tapscott, S. J. Tabrizi, A. H. Schapira, C. Kooperberg, et al.
Gene expression in Huntington's disease skeletal muscle: a potential biomarker
Hum. Mol. Genet., July 1, 2005; 14(13): 1863 - 1876.
[Abstract] [Full Text] [PDF]


Home page
Arch NeurolHome page
J. H. Friedman, M. E. Trieschmann, R. H. Myers, and H. H. Fernandez
Monozygotic Twins Discordant for Huntington Disease After 7 Years
Arch Neurol, June 1, 2005; 62(6): 995 - 997.
[Abstract] [Full Text] [PDF]


Home page
J. Neurol. Neurosurg. PsychiatryHome page
J M Hamilton, T Wolfson, G M Peavy, M W Jacobson, and J Corey-Bloom
Rate and correlates of weight change in Huntington's disease
J. Neurol. Neurosurg. Psychiatry, February 1, 2004; 75(2): 209 - 212.
[Abstract] [Full Text] [PDF]


Home page
NeurologyHome page
L. Djousse, B. Knowlton, L. A. Cupples, K. Marder, I. Shoulson, and R. H. Myers
Weight loss in early stage of Huntington's disease
Neurology, November 12, 2002; 59(9): 1325 - 1330.
[Abstract] [Full Text] [PDF]




HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Copyright © 1989 by The American Society for Nutrition