AJCN Tufts Nutrition Symposium, Boston Sept 24-26
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
QUICK SEARCH:   [advanced]


     

This Article
Right arrow Full Text (PDF)
Right arrow Purchase Article
Right arrow View Shopping Cart
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Adelekan, D. A.
Right arrow Articles by Thurnham, D. I.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Adelekan, D. A.
Right arrow Articles by Thurnham, D. I.
Agricola
Right arrow Articles by Adelekan, D. A.
Right arrow Articles by Thurnham, D. I.

American Journal of Clinical Nutrition, Vol 51, 453-456, Copyright © 1990 by The American Society for Clinical Nutrition, Inc

ORIGINAL RESEARCH COMMUNICATIONS

Plasma ferritin concentrations in anemic children: relative importance of malaria, riboflavin deficiency, and other infections

DA Adelekan and DI Thurnham
Department of Paediatrics and Child Health, Faculty of Health Sciences, Obafemi Awolowo University, Ile-Ife, Nigeria.

Anemia (hemoglobin less than 110 g/L) was documented in 36 children of both sexes aged 1-12 y who were divided into two groups: malaria and other infections. The control subjects were 10 children of similar age with no anemia and without any apparent infections. Plasma ferritin concentrations (median, range) were higher in the anemic patients (203 micrograms/L, 21-5000 micrograms/L) than in control children (52 micrograms/L, 25-239 micrograms/L) although ferritin concentrations in those with malaria were still within the normal range (99 micrograms/L, 21-205 micrograms/L). In the rest of the anemic group, five patients had plasma ferritin concentrations greater than 1000 micrograms/L. There was no difference in riboflavin status between control subjects and patients or between the two anemic groups. Severity of anemia was no different between the two anemic groups either. The data indicate that riboflavin deficiency makes no contribution to the infection- induced elevation in plasma ferritin and that the contribution of malaria is smaller than that of other unidentified factors.


This article has been cited by other articles:


Home page
 J. Nutr.Home page
A. Tomkins
Assessing Micronutrient Status in the Presence of Inflammation
J. Nutr., May 1, 2003; 133(5): 1649S - 1655.
[Abstract] [Full Text] [PDF]

Home page
 J. Nutr.Home page
R. J. Stoltzfus, H. M. Chwaya, M. Albonico, K. J. Schulze, L. Savioli, and J. M. Tielsch
Serum Ferritin, Erythrocyte Protoporphyrin and Hemoglobin Are Valid Indicators of Iron Status of School Children in a Malaria-Holoendemic Population
J. Nutr., February 1, 1997; 127(2): 293 - 298.
[Abstract] [Full Text]


HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Copyright © 1990 by The American Society for Nutrition