AJCN EB Program 2010 Early Registration
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
QUICK SEARCH:   [advanced]


     

This Article
Right arrow Full Text (PDF)
Right arrow Purchase Article
Right arrow View Shopping Cart
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Douglas, B. R.
Right arrow Articles by Lamers, C. B.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Douglas, B. R.
Right arrow Articles by Lamers, C. B.
Agricola
Right arrow Articles by Douglas, B. R.
Right arrow Articles by Lamers, C. B.

American Journal of Clinical Nutrition, Vol 52, 553-556, Copyright © 1990 by The American Society for Clinical Nutrition, Inc

ORIGINAL RESEARCH COMMUNICATIONS

Coffee stimulation of cholecystokinin release and gallbladder contraction in humans

BR Douglas, JB Jansen, RT Tham and CB Lamers
Department of Gastroenterology-Hepatology, University Hospital, Leiden, The Netherlands.

We studied the effect of the ingestion of 400 mL regular coffee on plasma cholecystokinin (CCK) concentrations and of 165 mL regular and decaffeinated coffee on plasma CCK and gallbladder contraction in six healthy regular coffee drinkers. Plasma CCK concentrations rose 3.3 +/- 0.4 pmol/L after 400 mL and 2.8 +/- 0.9 pmol/L after 165 mL regular coffee compared with 1.8 +/- 0.6 pmol/L after 165 mL decaffeinated coffee. These plasma CCK increments were greater than those after 400 and 165 mL of an isosmotic and isothermic sodium chloride solution (0.6 +/- 0.2 and 0.4 +/- 0.1 pmol/L, respectively). An average gallbladder contraction of 33 +/- 7% was observed after 165 mL regular coffee and 29 +/- 10% after 165 mL decaffeinated coffee, whereas after 165 mL sodium chloride the contraction was only 10 +/- 12%. We conclude that both regular coffee and decaffeinated coffee give rise to increments in plasma CCK and contractions of the gallbladder.


This article has been cited by other articles:


Home page
 J. Am. Coll. Nutr.Home page
A. Cuevas, J. F. Miquel, M. S. Reyes, S. Zanlungo, and F. Nervi
Diet as a Risk Factor for Cholesterol Gallstone Disease
J. Am. Coll. Nutr., June 1, 2004; 23(3): 187 - 196.
[Abstract] [Full Text] [PDF]

Home page
 Am J EpidemiolHome page
C. E. Ruhl and J. E. Everhart
Association of Coffee Consumption with Gallbladder Disease
Am. J. Epidemiol., December 1, 2000; 152(11): 1034 - 1038.
[Abstract] [Full Text] [PDF]

Home page
 JAMAHome page
M. F. Leitzmann, W. C. Willett, E. B. Rimm, M. J. Stampfer, D. Spiegelman, G. A. Colditz, and E. Giovannucci
A Prospective Study of Coffee Consumption and the Risk of Symptomatic Gallstone Disease in Men
JAMA, June 9, 1999; 281(22): 2106 - 2112.
[Abstract] [Full Text] [PDF]

Home page
 Arch Fam MedHome page
G. M. Etherton and M. S. Kochar
Coffee: Facts and Controversies
Arch Fam Med, March 1, 1993; 2(3): 317 - 322.
[Abstract] [PDF]


HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Copyright © 1990 by The American Society for Nutrition