AJCN Tufts Nutrition Symposium, Boston Sept 24-26
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
QUICK SEARCH:   [advanced]


     

This Article
Right arrow Full Text (PDF)
Right arrow Purchase Article
Right arrow View Shopping Cart
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Ostrom, K. M.
Right arrow Articles by Ferris, A. M.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Ostrom, K. M.
Right arrow Articles by Ferris, A. M.
Agricola
Right arrow Articles by Ostrom, K. M.
Right arrow Articles by Ferris, A. M.

American Journal of Clinical Nutrition, Vol 58, 49-53, Copyright © 1993 by The American Society for Clinical Nutrition, Inc

ORIGINAL RESEARCH COMMUNICATIONS

Prolactin concentrations in serum and milk of mothers with and without insulin-dependent diabetes mellitus

KM Ostrom and AM Ferris
Department of Nutritional Sciences, University of Connecticut, Storrs 06269-4017.

Diabetes may affect the secretion of prolactin, the principal lactogenic hormone. Because adequate amounts are critical to the establishment of lactation, we assessed the prolactin status of 33 women with insulin-dependent diabetes mellitus (IDDM), 33 women without diabetes, and 11 reference women participating in a study of lactation from 2 to 84 d postpartum. Circulating concentrations of serum prolactin declined temporally for all women and did not differ significantly among any of the groups. During the first postnatal week, milk immunoreactive prolactin concentrations were lower for women with IDDM than for control and reference women and the inverse relationship between lactose and milk prolactin, which was significant at day 2 postpartum for reference women, was delayed until day 14 postpartum for women with IDDM. Early breast-feeding activity, increased breast- feeding frequency, and good glycemic control enhance prolactin secretion and should be promoted during lactation in women with IDDM.


This article has been cited by other articles:


Home page
 J EndocrinolHome page
A. I Arroba, A. M Lechuga-Sancho, L. M Frago, J. Argente, and J. A Chowen
Increased apoptosis of lactotrophs in streptozotocin-induced diabetic rats is followed by increased proliferation.
J. Endocrinol., October 1, 2006; 191(1): 55 - 63.
[Abstract] [Full Text] [PDF]

Home page
 J. Nutr.Home page
D. I. Campbell, M. Elia, and P. G. Lunn
Growth Faltering in Rural Gambian Infants Is Associated with Impaired Small Intestinal Barrier Function, Leading to Endotoxemia and Systemic Inflammation
J. Nutr., May 1, 2003; 133(5): 1332 - 1338.
[Abstract] [Full Text] [PDF]

Home page
 J Hum LactHome page
K. L. Hoover, L. H. Barbalinardo, and M. P. Platia
Delayed Lactogenesis II Secondary to Gestational Ovarian Theca Lutein Cysts in Two Normal Singleton Pregnancies
J Hum Lact, August 1, 2002; 18(3): 264 - 268.
[Abstract] [PDF]

Home page
 J Hum LactHome page
J. Webster, K. Moore, G. Dip, A. McMullan, and B Nurs
Breastfeeding Outcomes for Women with Insulin Dependent Diabetes
J Hum Lact, September 1, 1995; 11(3): 195 - 200.
[Abstract] [PDF]


HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Copyright © 1993 by The American Society for Nutrition