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American Journal of Clinical Nutrition, Vol 8, 332-336, Copyright © 1960 by The American Society for Clinical Nutrition, Inc.
1 From the Heart Disease Control Unit, Mississippi State Board of Health, Jackson, Mississippi
It is obvious that coronary atherosclerosis in the human being is an end result of a number of causative factors. In contrast, the laboratory-induced lesion in the animal is most often caused by a single experimental technic. Thus, there is no common ground for comparison. There is a distinct possibility that an absolute or relative lipotropic deficiency is behind the disturbed lipoprotein metabolism, one of the atherogenetic factors in the form of the disease found in man. It is possible to demonstrate objectively the effect of lipotropic substances on lipoprotein metabolism.
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